development of IAA was considered possible because of oxidase deamination of NMT (Barker et al., 1980), but was later established to generally be also partly by immediate deamination by MAO (Barker et al., 1982). Pretreatment While using the MAO-I iproniazid in rat complete brain homogenates inhibited IAA formation by 83%, NMT and DMT-NO formation

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